Alzheimer’s Disease as a Condition of Many Subtypes and Contributing Causes

Neurodegeneration in late life is a very complex phenomenon, and its complexity strains against the nice neat clinical definitions of disease found in the textbooks. Different patients with Alzheimer’s disease can exhibit quite different mixes of various forms of pathology, developing at different paces and times: aggregates of amyloid-β, tau, and α-synuclein; vascular degeneration; markers of neuroinflammation; metabolic disruption similar to that of diabetes, and so forth. One case of Alzheimer’s might be different enough from another to require a different designation. Thus researchers talk about defining subtypes of Alzheimer’s disease, or that individual patients have Alzheimer’s that is exacerbated by a comorbidity arising from other neurodegenerative processes. Another way of looking at this is to categorize mechanisms that contribute to Alzheimer’s. To what degree

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